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Figure 2


Fig. 2. Comparison of the relaxation efficacy of 1 mmol l-1 H2S (as Na2S) and hypoxia (N2) on 10 µmol l-1 carbachol (CARB)-, 80 mmol l-1 KCl- and 80 mmol l-1 potassium acetate (KAc)-contracted trout urinary bladders (all N=8) and the effects of H2S application on a pre-existing N2 exposure and of N2 application on a pre-existing H2S exposure in pre-contracted bladders (all N=4). H2S (as NaHS) relaxation of CARB-contracted vessels is also shown. Values are means ± s.e.m. H2S was significantly less efficacious on KCl-contracted bladders than bladders pre-contracted with either CARB or KAc ({dagger}). Bladders relaxed with either H2S or N2 did not respond to the other (N2 or H2S, respectively), irrespective of the pre-contractile agonist (*). There was no difference in the relaxation produced by Na2S and NaHS.





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