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Fig. 6. Worms lacking the EAT-2 nicotinic receptor subunit initiate action potentials without MC-stimulated EPSPs. (A) EPGs from eat-2 mutants lack EPSPs and I-phase spikes because eat-2 mutants are defective in nicotinic neurotransmission from MC to the pharyngeal muscle. The ad465 allele of eat-2 contains an early stop codon. (B) EPGs from eat-2; cca-1 double mutants closely resemble those of eat-2 single mutants. E-phase spikes are marked with arrows.





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