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Fig. 4. Tension recordings showing the effect of acetylcholine (ACh) on the lateral
aorta with (A) or without (B) the presence of the NOS inhibitor
N
-nitro-L-arginine (L-NNA). The preparations were
pre-treated with L-NNA (10-4 mol l-1) for approximately
10 min before being constricted with endothelin-1 (ET-1; 10-8 mol
l-1). At the peak of vasoconstriction, acetylcholine (Ach;
10-5 mol l-1) was administered, which only caused a
marked vasodilation in the preparation without L-NNA (B) (N=5).
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