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Frontiers of hypoxia research: acute mountain sickness

Robert C. Roach* and Peter H. Hackett

New Mexico Resonance, Box 343, Montezuma, NM 87731, USA



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  		Fig. 1. Pathophysiology of acute mountain sickness (AMS). This schema emphasizes a role for blood–brain barrier opening (BBB), brain swelling and cerebrospinal compliance (CSC). Although highly speculative at present, new non-invasive and sensitive techniques will allow measurement of the variables necessary to evaluate this hypothesis (see text for more detail). pCap, cerebral capillary perfusion pressure; cCSF, cranial cerebrospinal fluid; ICP, intracranial pressure; CBV, cerebral blood volume; CBF, cerebral blood flow.

 


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  		Fig. 2. (A) Symptom scores for acute mountain sickness (AMS) severity and (B) arterial oxygen saturation measured by pulse oximetry (SaO2; %) during exercise in hypoxia. The different symbols in A represent the individual subjects in the two different trials. The solid squares in A represent the mean values ± S.E.M. for each trial. A drop in percentage SaO2 while exercising (*P<0.05 compared with the sedentary value) and a slight fluid retention (not shown) distinguished exercise (resulting in AMS) from rest (no AMS). Values are means + S.E.M. Adapted from Roach et al. (Roach et al., 2000).

 

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© The Company of Biologists Ltd 2001