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Fig. 8. (A) Effects of JSTX on cardiac acceleration induced by stimulation of the CA1 nerve at 3 Hz. Vertical lines show extracellularly recorded bursting impulses of the cardiac ganglion corresponding to heartbeats. The stimulation produced cardiac acceleration in normal saline (control; top trace). Small fine potentials during the bars in the records are stimulus artifacts. Application of JSTX (10–5 mol l–1) blocked the CA1-induced acceleration at 15 min after the onset of the application (middle trace). The stimulation of the CA1 nerve at 3 h after washing out JSTX induced cardiac acceleration (wash; bottom trace). The bar shows a period of repetitive stimulation. (B) Effects of JSTX on cardiac acceleration induced by stimulation of the CA2 nerve at 10 Hz. Application of JSTX blocked the CA2 induced acceleration at 30 min after the onset of the application. The stimulation of the CA2 nerve at 1 h after washing out JSTX induced cardiac acceleration. Small fine potentials during the bars in the records are stimulus artifacts.